Psychology

Cannon Bard Theory

The Cannon-Bard theory of emotion proposes that emotions and physiological responses occur simultaneously, rather than one causing the other. According to this theory, an emotional stimulus triggers both the experience of an emotion and the physiological response independently. This challenges the earlier James-Lange theory, which suggested that physiological responses lead to the experience of emotion.

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3 Key excerpts on "Cannon Bard Theory"

  • Principles Of Biopsychology
  • If peripheral arousal is sufficient to produce emotions, then simply taking physical exercise should be effective. But running up the stairs is not, by and large, an emotional experience.
  • Emotion: James-Lange Peripheral Theory (panel A).
    Emotion: Cannon’s Central Theory (panel B).
    In fact some of these criticisms have themselves fallen by the wayside. Hohmann (1966), studying a group of 25 patients with spinal cord damage, reported that they did experience reductions in feelings of anger, fear, and sex, in line with predictions from the James-Lange model.
    Cannon’s alternative model gave a major role to the thalamus in the diencephalon. Based mainly on research with animals (see last section), much of it by Bard and so referred to as the Cannon-Bard model, Cannon proposed that sensory input from the environment passes to the cortex, which, after processing it, passes the results to the thalamus. Thalamic activity produces the experience of emotion, while connections from the thalamus to the hypothalamus are responsible for the eventual behavioural expression of emotional states.
    In support of Cannon’s position, Maranon (1924) had systematically manipulated arousal and failed to affect emotion. He used the drug adrenaline (known as epinephrine in the USA); this is also the hormone released from the medulla of the adrenal gland which stimulates and sustains peripheral arousal. Maranon argued that even if a state of arousal was produced artificially, by the injection of adrenaline, the James-Lange model would still predict that subjects should feel emotions.
    Of his 210 subjects, 71% reported only the physical consequences of adrenaline—such as increased heart-rate, dry mouth etc—whereas 29% reported “as if” emotions, i.e. not genuine experiences, but as if they were afraid or angry etc. Thus the James-Lange model was not supported.
    The next major contribution to the debate on the role of peripheral arousal in emotion was a study that has become one of the most famous in the whole of psychology. In 1962 Schacter and Singer attempted to resolve the James-Lange / Cannon debate in a single experiment. They were convinced that although peripheral arousal was necessary for emotions to occur, it needed to be combined with a cognitive appraisal of the situation. This appraisal allows the subject to interpret the bodily arousal appropriately; if the situation involves a raging bull, you interpret the arousal as fear, whereas if it involves someone recording over your favourite video you would interpret it as anger. They point out that in Maranon’s study the subjects had an obvious explanation for their state of arousal, as they knew that they had been injected with adrenaline. Their cognitive appraisal would not involve emotion-inducing stimuli, so they did not feel emotional.
  • Companion Encyclopedia of Psychology
    • Andrew M. Colman(Author)
    • 2018(Publication Date)
    • Routledge
      (Publisher)
    My thesis, on the contrary, is that the bodily changes follow directly the perception of the exciting fact, and that our feeling of the same changes as they occur IS the emotion. Common sense says, we lose our fortune, are sorry and weep; we meet a bear, are frightened and run; we are insulted by a rival, are angry and strike. The hypothesis here to be defended says that this order of sequence is incorrect... and that the more rational statement is that we feel sorry because we cry, angry because we strike, afraid because we tremble, and not that we cry, strike, or tremble because we are sorry, angry, fearful, as the case may be. (p. 449, emphasis in original) James (1898) argued that the differences between emotions are a direct result of the different patterns of physiological response associated with them. According to James, seeing something frightening instinctively triggers a whole set of reactions in our bodies. The particular pattern of these reactions is felt by us consciously and experienced as the particular emotion of fear. For example, our feelings of fear might be based on the feeling of raised blood pressure, tightening of our muscles, a certain sensation in our guts. Figure 1 Sequences of emotion: early theories One of the main problems with James's theory, as pointed out by Cannon (1927), is that the physiological changes that accompany a wide variety of emotions are actually very similar, and certainly not distinct enough to differentiate widely contrasting subjective states such as euphoria and anger. Cannon's alternative theory represented a slight return towards the commonsense view of emotion. Cannon's idea was that emotional encounters directly triggered a central brain process in the thalamus, which had two simultaneous and independent outputs, one to the arousal system which prepared the body to cope with the emergency, and the second to the cortex where the conscious experience of the emotion was registered (see Figure 1c)
  • Fundamentals of Psychology
    There is a bewildering variety of theories of emotion. They differ considerably because of the different aims of the theorists concerned. Some theorists have viewed emotion mainly from a physiological perspective, whereas others emphasize the cognitive processes associated with emotion. Still other theorists have provided an overall account of the relationships among the cognitive, physiological, and behavioral systems.
    The first major theory of emotion was put forward independently by William James in the United States and Carl Lange in Denmark in the mid-1880s. This could explain why the theory is generally known as the James–Lange theory. According to this theory, three successive stages are involved in producing emotion:
    1. There is an emotional stimulus (e.g., a car comes rapidly towards you as you cross the road).
    2. This produces bodily changes (e.g., arousal in the autonomic nervous system).
    3. Feedback from the bodily changes leads to the experience of emotion (e.g., fear or anxiety).
    Common sense might suggest that (2) and (3) are in the wrong order. For example, James gave this example of the predicted sequence according to the theory: “I see a bear, I run away, I feel afraid” (see the figure below). It seems more likely that the sequence would be as follows: “I see a bear, I feel afraid, I run away.”
    There is some mileage in the James–Lange theory. For example, consider patients with panic disorder experiencing a panic attack. The key reason they experience extremely high levels of anxiety is because they catastrophically misinterpret their own bodily symptoms (e.g., interpreting a fast heart rate as indicating an imminent heart attack) (Clark, 1986 ). However, the theory assumes mistakenly that each emotion is associated with its own specific pattern of physiological activity. In addition, evidence from patients with damage to the spinal cord is not favorable to the theory. These patients have greatly restricted awareness of their own physiological symptoms, and so should have a large reduction in their emotional experience. However, Bermond, Nieuwenhuyse, Fasotti, and Schwerman (1991) found that most of their patients with spinal damage reported increased intensity of emotions. Cobos, Sánchez, Pérez, and Vila (2004) found that patients with spinal cord injuries reported that their current emotional experiences across several emotions were at least as great as prior to injury. Thus, strong feedback from bodily changes is not
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